lfredrick123

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About lfredrick123

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  • Service Connected Disability
    10%
  • Branch of Service
    Army

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  1. ICD 9 and SCT are codes used in the insurance industry to identify a specific diagnosis. The Systematized Nomenclature of Medicine — Clinical Terms (SNOMED-CT) was created by the College of American Pathologists (CAP) to represent medical terminology in electronic health records (EHRs) For many years providers have been doing a good job of summarizing their patients’ current and relevant medical conditions on a “problem list”. Typically this list is located within the first page of a patient’s chart, ideally enabling the medical provider to quickly assess the current and past medical issues of the patient. While the intent is clear, the methodology is not – many providers still using paper charts may use acronyms to express a clinical condition (e.g. MS or AA) or they may not add the date of the diagnosis and/or its resolution. For those providers who utilize EMRs (electronic medical records) the problem may be more complex due to the lack of interoperability between different EMR systems. Enter Meaningful Use Stage 2 and SNOMED. Stage 2 Meaningful Use criteria expands upon the Stage 1 requirements to further improve and utilize healthcare IT and EMRs to provide consistent, collaborative care among different provider groups for any given patient. This means that these electronic systems need to talk to each other and more importantly they need to understand each other. The only way for them to reach this understanding is to speak a common language. Stage 2 of Meaningful Use has defined this language as SNOMED-CT – specifically for the problem list within a patient’s chart. This is an acronym for Systematized Nomenclature of Medicine – Clinical Terminology. It is recognized throughout the US and internationally, and it is available at no cost through the National Library of Medicine. Using SNOMED-CT enables providers and electronic medical records to communicate in a common language, thus increasing the quality of patient care across many different provider specialties. ICD-9 was and is used as a diagnosis and procedure coding system. Again specifically outlining the diagnosis and the process followed for a precedure performed. Its now an old system and is supposed to have been upgraded to ICD-10 this last year,
  2. Same thing they did with Vietnam and Cold War Vets. The only thing you can do is fight and beat them at their own games.
  3. Hi Mike...we just went through the fiduciary thing ourselves. Not to worry, if you have officially nominated your GF, she has a clean background and credit report. You should call ebenefits and speak to someone there to help you. They can check on where the awards are and if you have started the fiduciary process. The field examiner will come out and check for safety and soundness, and then develop a budget for you if you dont have one already. We did and I gave it to him. The process takes awhile but is not that difficult really, just nerve wracking. You do not need to be service connected to get medical help ( you already are at 70%) just go to the VA hospital and get your id and an enrollment done. Any veteran with a honorable discharge can go there. They can help get you on track but make sure you have your GF with you as an advocate. It took us a short time but we got in. As for the $ your owed you need to call to track that down. Ebenefits should have ypur information under the payments tab and if not there then look at your profile for what account you have set up if any. I would call because they can check right away. Most of all get the help you need, you can call the social workers there and they can helo you get on your feet. I will say some extra prayers tonight you get this quickly. Its not insurmountable, just arduous.
  4. Must be contageous as we have it too. Wonder if Obsessive Compulsive Disorder would work? (Ha Ha) Really do not understand why it would be so difficult to post an updated status once every 6 months. I know they have alot going on but really its not helping their volumes at all to force people to call in for it.
  5. Hi Berta,

    Thank you for all you do for Veterans, I am wondering if you could address this. I am helping my husband with his claims and pending appeals. He has a multitude of issues following a debilitating hemmorhagic stroke in 2008. We got his service records over 5 different requests that provided his service medical treatement records and service records, I had an IMO done for him with his medical records and his personal records. The STR show he had an elevate BP at entry of 140/90 which was very high for someone 22resting. After ecercise it was 148/94. it continured over the years and for any years the docs kepot telling him diet and exercise which he did do all the time. In fact he was riding his bike as usual, when he collapsed. His BP has gone as high as 193/100 or more on occassions, In 2004 long before the stroke he was diagnosed with COPD, even though he never smoked, He has gone 2 times in service to the dispensary but dont know what for, since there are no records, He says because the pesticide fumes made him sick that they were spraying in the facilities and around the base. I see a Dispensary notation but no records in his C file. Also no records on injections they gave, Just that he was there. His dental records are there, and his eye glass records but not the 2 visits that are showing. Also found a record of his hand injury and sutures. At discharge the record shows nothing other than a lower BP reading,and 0's accross the audiogram( he says he never had an exam) and to boot I found in the C file a lab document showing a retake of a urine with a Creatinine of 1.4 (Kidney disease?) I asked the Army for records they insisted they had none on pesticides. I did my own digging found a retired COL. who was Chief of Environmental Command there and he told me they were spraying in 1962-1963 Diazinon. Mallathion,Chlordane, DDT, Lindane, Nicotine Sulfate, 2,4,5,-T along with 2,4,D. He put that in writing, I also searched further found out that the baserecieved a shipment of Phosgene and it was just before his arrival. They had a chemical weapons training field they had used land mines filled with mustard in the 40s that they used for training, And then I got the Industrial Hygiene Surveys that ran from 1956.57.58.59.60.61.63.63 all which confirmed heavy carbon monoxide levels in training facilities (and in tank exhaust) along with none other IH concern about the pesticides which were sprayed by 5-8 men, 8hrs per day from April until October, IMO doc issued the following report: I could not get the name to redact so I have just ssent it to you in whole, If you can change it there I would appreciate it. My system is old, and my scanner just files it as is. At any rate I am wondering your opinion on this case now, All have been sent to VADr Ellis MD Nexus Letter Nov 20 2015 (3) - Copy.jpgDr Ellis MD Nexus Letter Nov 20 2015 (3) - Copy.jpgDr Ellis MD Nexus Letter Nov 20 2015 (4) - Copy.jpgDr Ellis MD Nexus Letter Nov 20 2015 (4) - Copy.jpgDr Ellis MD Nexus Letter Nov 20 2015 (5) - Copy.jpg

     

    Dr Ellis MD Nexus Letter Nov 20 2015 (2) - Copy.jpg

    1. Berta

      Berta

      I don't feel Dr. Ellis's  medical rationale is strong enough for some of these issues.

      Did he fully opine on the MRI of the CVA?

      Did he rule out ischemic heart disease as potential cause of stroke?

      Or transcient ischemia due to diabetes causing stroke?

      In case the stroke was not hemorrhage at all but instead ischemic ....a mistake VA made when they initially diagnosed my husband's stroke.

      HBP can contribute to stroke as my recent CUE 1151 award reveals. But I don't think that evidence would help you.

      I think you should post in the main forum where others can also opine on this. I hardly ever read anything in my profile,except to find my last posts.

      We would need to see what his SC ratings are now and what for, as well as a  copy of any denials he received that are being appealed.The C file, name ,address ect. can be covered prior to scanning and attaching to a post in the main forum.

      A copy of the C & P exams would be good too. in the public forum....where more can opine on all this.

       

    2. lfredrick123

      lfredrick123

      Thanks Berta, I will post and make sure the evidence supports this info.

  6. Dont know if this will help, but husband served in a tank squadron as a turret artillery repairman. Tanks were known to have heavy Carbon Dioxide from the diesel fuels in the turret and in the smoke and obliterants. We were digging for military evidence and found industrial hygiene surveys done at the base he was at that had multiple years of reporting that showed excessive levels of Carbon Monoxide in the training areas and heavy pollution levels. You might check to see if there were any surveys done at your particular base that would show it as a health hazard, and if they were doing anything at all to help. In husbands case he has COPD from it (never smoked himself) and severe lung problems now thanks to exposures and an eventual stroke. At any rate I found a report from Dr Grace Ziem on Hydrocarvons, fuels exposures. Here it is. If you will look her info up Dr. Grace Ziem MD she is an Occupational doc who worked as a consultant to VA and there may be more info on your cancer problems in her materials. Here is what I found: COMBUSTION PRODUCTS Being near busy traffic or vehicle exhaust causes exposure that affects lung function. 1 This is worse for those with longer exposure, such as house near traffic.1 Very small vehicle particles can easily enter an indoor environment. 1 Exposure to diesel exhaust causes lung inflammation even in healthy people. 2 Risk is greater with longer exposure as well as higher intensity. Even higher risk occurs with indoor exposure to idling vehicles.3 DANGER BELOW EXPOSURE LIMITS Harmful health effects occur from combustion product particles even at below government and commonly used exposure levels. 4 Particulate pollutants (such as combustion products) cause and exacerbate illness at levels below EPA and WHO guidelines.5 There is a direct dose-response relationship between levels of combustion particle exposure and death rate. 4 Recent scientific research shows body damage at very low levels of carbon monoxide, suggesting there is no known safe level of carbon monoxide exposure.6 HAZARDOUS GASES Combustion gases contain irritants7 8 9 and toxins.10 11 Combustion products of fuels (oil, gasoline, diesel, propane, etc.) contain the irritant oxides of nitrogen and for most fuels, oxides of sulphur. Repeated modest and even "tolerable" level irritant exposure,8 or higher level single8 or repeated9 irritant exposure can cause permanent or long term reactive airway disease rendering the individual with long-standing heightened susceptibility to exacerbating symptoms from future irritant exposures.7 9 Combustion products such as vehicle exhaust and smoke release benzene, a potent cancer agent, and these exposures increase cancer risk.10 Combustion products also contain carbon monoxide, which can cause sensitization and affect memory and learning.11 Carbon monoxide is toxic to brain/nerve cells, the heart, and other body muscle.6 Carbon monoxide exposure can cause long term neurologic damage.6 Chemical exposure can also interfere with detoxification. 11 LUNG DAMAGE FROM COMBUSTION PARTICLES Combustion particles when inhaled can cause allergic effects and other chronic respiratory damage.3 12 13 14 Combustion particles can accumulate in the lungs. 15 Gases, vapors and other air pollutants cling to particles and the particles then carry these substances into the lungs.16 They persist longer, because particles are harder to clear from the lungs. Very small particles cause lung inflammation, damage lung cells, and form lipid peroxides in lung tissue. They can also enter the body through the lungs and/or cause lung scarring. 17 Combustion Products Page 2 of 5 Fine particulates deposit in the respiratory tract.14 Smaller particles (under 2.5 microns) deposit in the deep lung sacs (alveoli).14 They cause inflammation that makes lungs more permeable (to toxins, other particles, etc).14 Fine particles can act as a physical stressor, increasing norepinephrine (adrenalin) and adrenal cortisol (body stress hormone) levels.14 EXPOSURE SOURCES All combustion releases carbon monoxide, and most combustion releases particles as well. Burning landfills and industrial releases are major sources. Vehicles are a problem especially with frequent/repeated and/or heavy traffic exposure, gas or kerosene space heaters, gas appliances and tobacco sources release indoor carbon monoxide.6 DIESEL Exposure to diesel exhaust causes eye and respiratory irritation and can lead to chronic respiratory damage.3 These very small particles entering the blood stream can impair normal function of the autonomic nervous system.17 Repeated, chronic diesel exhaust exposure can also cause brain damage with documented impairment in memory and other cognitive functions, as well as impaired balance, reaction time and other neurophysiologic functions.3 HEART DAMAGE Particles can cause changes in EKG (electrocardiogram) tests showing (reduced blood/oxygen supply and/or inflammation.18 Exposure to combustion particles and gases cause excess cardiovascular disease risk 19 20 21 and risk of death from stroke and other causes.21 HOW COMBUSTION PARTICLES CAUSE HARM When fine chemical particles are breathed in, they can pass into the blood stream and be distributed to many other body organs and cells.20 Chemical particles in those other locations also cause inflammation in those locations. They cause immune changes. They also cause toxicity20 and increased need for antioxidants due to formation of tissue-damaging substances called free radicals.20 Bigger particles breathed can penetrate and be deposited in the larynx (voice box), trachea and larger airways14 causing inflammation.14 22 Combustion particles impair lung function.23 Ultrafine particles in large numbers are present in vehicle emission, worse in diesel. These particles have a high ability to attach to lung sacs (alveoli), cause inflammation. They also enter the blood stream16 and have a large surface area to absorb gases and vapors.16 They thus carry other vapors into the body. COMBUSTION PRODUCT EXPOSURE DEPLETES ESSENTIAL ANTIOXIDANTS AND NUTRIENTS Carbon monoxide exposure11 24 25 26 can produce excess nitric oxide in the body 11 24 25 and NMDA activation.25 27 Both of these lead to inflammation.26 28 29 30 31 Particles also cause inflammation and increased nitric oxide. 22 32 These changes all deplete nutrients and require nutrients for repair.28 33 34 35 36 37 38 39 40 41 42 43 44 45 46 47 48 49 50 51 52 53 54 Combustion Products Page 3 of 5 This damage can be reduced by treatment with glutathione.12 Fine combustion and other particles can cause increase in the respiratory inflammation marker, exhaled nitric oxide.22 Excess nitric oxide depletes cobalamin (B12) and needs hydroxocobalamine as a scavenger. 36 37 OTHER BODY DAMAGE They can cause damage to genetic material (DNA).10 This is measured by a substance called 8-hydroxy-2-deoxyguanosine.16 This type of DNA damage could lead to increased cardiovascular and pulmonary disease, risk of mutations and cancer.16 Diesel exhaust increases lung cancer.55 They also damage essential lipids,16 causing damage to cell membranes and membranes around internal structures inside cells. Examples of these vital structures are ribosomes making proteins, mitochondria producing energy, etc.). They damage myelin in the brain and nervous system. Exposure to vehicle traffic exhaust significantly increases body exposure to these particles,16 and increased DNA damage can be measured after such exposure.16 It is worse with heavier traffic16 e.g., commuter, highway traffic, etc.). Other studies confirm a correlation between DNA damage and exposure to small particles. Inhaled ultrafine particles can penetrate through the lung and within an hour are able to penetrate cells and affect energy-generating mitochondria and other structures within body cells.16 56 Carbon monoxide can also cause inflammation of blood vessel linings.57 This can impair oxygen supply to the brain, heart and other organs. BURNING SYNTHETICS Persons exposed to combustion products from flame-retardants in plastics, electronics, fabrics and other materials can develop permanent brain and neurologic damage.58 Deca is the most widely used flame retardant and during combustion and other exposure breaks down to brominated compounds. These persist in the body for decades and are banned in the European Union and California.)58 1 D Sugiri, etal, “The influence of large-scale airborne particle decline and traffic-related exposure on children’s lung function,” Env Health Persp 114:282-288, 2006. 2 S Dubowsky Adar, etal, “Ambient and microenvironmental particles and exhaled nitric oxide before and after a group bus trip”, Env Health Persp 115: 507-512, 2007. 3 KH Kilburn “Effects of Diesel exhaust on neurobehavioral and pulmonary functions,” Archiv Env Health, 55: 11-14, 2000. 4 E Samoli, etal, “Particulate air pollution and mortality: findings from 20 US cities, N Eng J Med 343: 1742-1757. 5 SV Glinianaia etal, “Does particulate air pollution contribute to infant death? A Systematic Review,” Env Health Persp 112: 1365-1370, 2004. 6 E Samoli, etal “ Short-term effects of carbon monoxide on mortality: An analysis within the APHEA project”, Env Health Persp 115: 1578-1583, 2007. 7 HM Kipen etal., "Asthma experience in an occupational medicine clinic. Low dose reactive airway dysfunction syndrome", J Occup Med. 36: 1133-1137, 1994. 8 SM Brooks etal, "Reactive airway dysfunction syndrome", J Occup. Med. 27: 473-476, 1985. 9 M Tarbo and I Broder, “Irritant-induced occupational asthma”, Chest 96: 297-300, 1989. 10 MN Mead “A backpack’s worth of data: Elevated teen cancer risks linked to air pollution”, Env Health Persp, 114: A601, 2006. 11 NH Prahhaker and RS Fitzgerald, “Carbon monoxide: From tool to neurotransmitter”, CRC Press, 1996. 12 JE Sharmon etal., "Exposure to automotive pollution increases plasma susceptibility to oxidation", Arch Env Health 57: 536-540, 2002. 13 FD Gilliland etal., “Effect of glutathione-S-transferase M1 and P1 genotypes on xenobiotic enhancement of allergic responses: randomized, placebo controlled crossover study”, The Lancet 363: 119-124, 2004. 14 MP Sirivelu, etal “Activation of the stress axis and neurochemical alterations in specific brain areas by concentrated ambient particle exposure with concomitant allergic airway disease”, Env Health Persp 114: 870-874. Combustion Products Page 4 of 5 15 A Penn etal, “Combustion-derived ultrafine particles transport organic toxicants to target respiratory cells”, Env Health Persp 113: 953-963, 2005. 16 P Vinzents etal, “Personal exposure to ultrafine particles and oxidative DNA damage”, Env Health Persp 13: 1485-1490, 2005. 17 C Chang-Chuan etal, “Personal exposure to submicrometer particles and heart rate variability in human subjects”, Env Health Persp 112: 1063-1067, 2004. 18 DR Gold, etal, “Air pollution and ST-segment depression in elderly subjects”, Env Health Persp 113: 883-887, 2005. 19 LH Chen, etal “The association between fatal coronary heart disease and ambient particulate air pollution: are females at greater risk?” Env Health Persp 113: 1723-1729, 2005. 20 TR Nurkiewicz, etal, “Systemic microvascular dysfunction and inflammation after pulmonary particulate matter exposure”, Env Health Persp 114: 412-419, 2006. 21 A Zanobetti and J Schwartz, “Particulate air pollution, progression, and survival after myocardial infarction”, Env Health Persp, 115: 769-775, 2007. 22 F Therese, etal Exhaled nitric oxide in children with asthma and short-term PM 2,5 exposure in Seattle”, Env Health Persp 113: 1791-1794, 2005. 23 SF Suglia, etal, “Association between traffic-related black carbon exposure and lung function among urban women”, Env Health Persp 116: 1333-1337, 2008. 24 SR Thom etal “Neuronal nitric oxide synthase and n-methyl-d-aspartate neurons in experimental carbon monoxide poisoning”, Toxicol Applied Pharmacol 194: 280-295, 2004. 25 RE Lenga, Ed., Sigma-Aldrich Library of Chemical Safety Data. Sigma-Aldrich Corp, 1988. 26 R Dales, etal, “The influence of living near roadways on spirometry and exhaled nitric oxide in elementary school children” Env Health Persp 116: 1423-1428, 2008. 27 WA Pryor and G Squadrito, “The chemistry of peroxynitrite: a product of the reaction of nitric oxide and superoxide”, Am J Physiol 268:L 699-L, 722, 1995. 28 M Lafon-Cazal etal, “Nitric oxide, superoxide and peroxynitrite: putative mediation of NMDA-induced cell death in cerebellar cells”, Neuropharmacology 32: 1259-1266, 1999. 29 JS Beckman, “The double edged role of nitric oxide in brain function and superoxide-mediated injury”, J Dev Physiol 15: 53-59, 1991. 30 M Lafon-Cazal etal, “NMDA-dependent superoxide production and neurotoxicity”, Nature 364: 535-537, 1993. 31 JT Coyle, P Puttfarken, “Oxidative stress glutamate and neuro generative disorders”, Science 262: 689-695, 1993. 32 JE Haley, etal, “Evidence for spinal N-methyl-D-aspartate receptor involvement in prolonged chemical nociception in the rat”, Brain Res 518: 218-226, 1990. 33 S Moncada and J Bolanos “Nitric oxide cell bioenergetics and neurodegeneration”, J Neurochem, 97: 1676-1689, 2006. 34 JS Beckman and JP Crow, “Pathologic implications of nitric oxide, superoxide and peroxynitrite formation”, Biochem Soc Trans 21:330-333, 1993. 35 M Wolak, etal, “Kinetics and mechanism of the reversible binding of nitric oxide to reduced cobalamin B(12r) (Cob(II) alamin)” J Am Chem Soc 123: 9780-91, 2001. 36 C Zheng, etal, “Electrochemical and spectral studies of the reactions of aquacobalamin with nitric oxide and nitric ion”, Inorgan Chem, 41: 2548-2555, 2002. 37 W B Slot, etal, “Normalization of plasma vitamin B12 by intranasal hydroxycobalamine in vitamin B12 deficient patients”, Gastroenterology 113: 430-433, 1997. 38 O Stanger, M Weger, Interactions of homocysteine, nitric oxide, folate and radicals in the progressively damaged epithelium”, Clin Chem Lab Med 41: 1444-1454, 2003. 39 CD Klassen etal, Editors, Cassarett and Doulls Toxicology: The Basic Science of Poisons, McGraw Hill, New York, NY 2001. 40 W Watson, etal, “ A new role for glutathione: Protection of vitamin B12 from depletion by Xenobiotics”, Chem Res Toxicol 17: 1562-1567, 2004. 41 S Oja etal, Modulation of glutamate receptor functions by glutathione” Neurochemistry International 37: 299-306, 2000. 42 H Sprince etal, “Comparison of protection by L-ascorbic acid, L-cysteine, formaldehyde toxicity”, agents and actions 9: 407-414, 1979. 43 LJ Machlin and A Bendich “Free radical damage: protective role of antioxidant nutrients”, FASEB J 6: 441-445, 1987. 44 T Konrad etal, “Alpha-lipoic acid decreases serum lactate and pyruate and improves glucose effectiveness in lean and obese patients with type 2 diabetes”, Diabetes Care 22: 280-287, 1999. 45 S Hustad etal, “Riboflavin, flavin mononucleotide, flavin adenine dinucleotide in human plasma and erythrocytes at baseline and after low-dose riboflavin supplementation”, Clin Chem 48; 1571-1577, 2002. 46 A Prentice, C Bates, “A biochemical evaluation of the erythrocyte glutathione reductase (EC1.6.4.2) test for riboflavin status. Dose response relationships in chronic marginal deficiency”, Brit J Nutr 45: 53-65, 1981. 47 G Ziem, “Endocrine Changes in Patients with Chronic Illness Following Chemical Overexposure”, Invited presentation at Conference on Chemical Injury, Fairfax, VA, October 3, 2003. 48 M Jones, “Chronic neuropathic pain: pharmacological in the new millennium”, Internat J Pharmaceutical Compounding, Jan-Feb 2000. 49 S Christen etal, “Gamma-tocopherol traps mutagenic electrophiles such as NOx and complements alpha-tocopherol: Physiologic implications” Proc Natl Acad Sci 94: 3217-3222, 1997. 50 K Wagner etal, “Gamma-tocopherol-an underestimated vitamin” J Nutr 122: 2440-2446, 1992. Combustion Products Page 5 of 5 51 JO Moskaug, etal, “Dietary Polyphenols and Health: Proceedings of the 1st International Conference on Polyphenols and Health”, Am J Clin Nutr 81: 277S-283S, 2005. 52 N Kocak-Toker, etal, “Peroxynitrite induced decrease in Na+ , K+ -ATPase activity is restored by taurine.” World Journal of Gastroenterology. 11: 3554-3557, 2005. 53 T Yokozawa etal, “(-)-Epigallocatechin 3-0-gallate ameliorates the damage related to peroxynitrite production by mechanisms distinct from other free radical inhibitors”, Xenobiotica 33: 913-25, 2003. 54 AY Sun, YM Chen, “Oxidative stress and neurodegenerative disorders”, J Biomed Sci, 5: 401-14, 1998. 55 E Garshick, etal, “Lung cancer and vehicle exhaust in trucking industry workers”, Env Health Persp 116: 1327-1332, 2008. 56 B Weinhold “Particles in practice: how ultrafines disseminate in the body”, Env Health Persp 113: 758. 57 MJ Campen, “A comparison of vascular effects from complex and individual air pollutants indicates a role for monoxide gases and volatile hydrocarbons”, Env Health Persp, 118: 921-927, 2010. 58 “Labor drug sensitizes brain to pesticide injury”, Our Toxic Times, December 2004, Duke University Press Release, March 30, 2004. Hope this helps
  7. We sent in a timely NOD and Request for DRO Review. That is all that was available on the form, It is written Decision Review Officer (DRO) Review Process or Traditional Appellate Review Process as the options. We chose the DRO Review process. Hopefully this will get us to a hearing on the matter rather than them just reviewing the decision. Any suggestions? We are beyond the one year time period now. Or should we send a certified letter to them confirming the request and DRO Hearing.
  8. Dr Ellis did my husbands as well, I sent him the medical records, hospital summaries, consults, discharge reports, and proof of exposures as well. We got our IMO back in about 2 weeks time and it was very thorough. We sent it on to VA but remains to be seen if they will ever review it. They are very slow in dealing with anything.
  9. We are waiting on a sleep apnea claim as well. Hubby had the study and clearly because he is a CO2 retainer and has hypoxia on top, it has caused Respiratory failure. He no long sleeps just 4 hrs but a full 9 , his snoriing had subsided along with the night terrors, His heart has improved and he if feeling better as well. He has a tracheostomy and so his maching attaches diredtly to that. Had to juno through mounds of hoops to get it however and many questions from Drs about it. I actually had to go with him into a trach hospotal for 3 weeks  with him to train in its use. Its been a big help.

    1. EODCMC

      EODCMC

      Wow, my heart goes out to you and your husband. I wish you well.

    2. EODCMC

      EODCMC

      BTW, my wife is hailing the CPAP. I have not woken her once since adjusting to it. For that reason alone, it is worth it. Had I know, I would have started years ago. 

  10. This is what we went through too. Eventually I figured we would be waiting for ions, so we sent a certified return reciept FOIA and Privacy Act request to them again, but this time also sent a letter to the General Counsel's office and told them we would be complying with the FOIA and Privacy Act Laws and if necessary we would file in Federal Court to get the C file, We got the Cfile within a few days, Its rediculous when you go months without even an acknowledgement. And clearly I felt like they were deliberately doing this to interfere with my husbands ability to get an independent medical exam, because you need the files to give to them.I looked the VA rules on FOIA requests and followed the law as it mandated,
  11. We waited almost a year on my husbands file and according to them when we called it could take up to three years. Since I had sent them the request cerified mail and a Privacy Act Request originallym I sent a 2nd request certified just prior to the 1 year mark. When they failed to answer I sent a final request through the Office of the General Counsel and got it in less than a week later, Know they are busy, and no doubt a thankless job, but veterans are waitiing for help, and must have their documents.
  12. First of all request your entire claims file including all of your VA and Service Treatment records from the date of your entry into the service and up to present date. You need to include a photocopy of your driver's license and the necessary VA form you need by going to the VA medical center for your area. You will see a patient portal and when you click on it, it. Will take you to how to request medical records. Fill it out and sign it and send with the. ID. It would be better if you could go in person but if not send it to the health information officer at the med center,make a copy of the request and letter and send it to the claims processing center too. These have to go certified return receipt. Keep a copy for yourself too for your file. This will request your health records they have at VA. This is all part of Hippa regulations and the Privacy act. If you don't get it in 30 days, send again a 2nd request certified. If you're still waiting then call the health information officer personally. Now for your personal records I would call Social Security and ask for a complete set of your file undr a GOIA and Privacy Act request. They should have all your prior records. Generally the costs are less than going to every doctor. If you need help the Vet centers. Can help.
  13. Also just want to wish you all a Very Merry Christmas and a Wonderful New Years. And if your not Chrisitan, then a Holiday of your choosing that is bright, loving and fullfilled from our hearts to yours,
  14. Interesting stuff here. We recently asked for a DRO and updated new evidence plus an IMO that gave a nexus on the stroke, connected to hypertension that was clearly in the service record. We also gave them uploaded evidence we got from 2 witnesses, and on top of that mounds of evidence from our own doctors since. We had asked for the C file in May 2015, Got nothing Yet, Sent several requests for 2nd and 3rd time to no avail. Finally a lawyer told me to send a final request certified mail to the Central VA Office, and if they did not respond in the 20 days, then file an appeal with the General Counsel about the issue. We did exactly that. If we dont get the response then we will file the sppeal in Federal Distric Court to compel it. There is no reason it should not have been done. And no reason access to his own records not provided. We had enough info from our old copies of Service Records to give to the IMO doctor, along with the VA Health records. I didnt have one of the independent C and P Exams and told the doctor and showed him the multiple requests. He was able to review what I did have, and he wrote the nexus, So we will see when they produce the C file. If they dont then they will have failed in their duty to assist in this case.
  15. We used Dr John Ellis MD WHERE ARE THEY: Oklahoma City OK NAME: Dr John Ellis WHAT IS THEIR SPECIALTY (or what you were seen for):- He does Medical Lar and Forensic Medicine, he is an expert in Occupational Medicine, and he teaches other medical progessionals about Forensic medicine , His Curriculum Vitae is extensinve. He is Board Certified, and his practice is primarily Occupational Medicine. COST:$650 CONTACT: 5100 N Brookline Ave # 465, Oklahoma City, OK 73112 (405) 917-5336 REVIEW (keep the Yes or No, based on whats correct) Did they review your entire file (YES) (NO) -- Yes Did they provide a well written DBQ/IMO/IME (YES) (NO) - Yes Were they familiar with VA Claims (38 cfr, M21, etc) ? (YES) (NO) -- Yes Would you classify them as "Vet Friendly" (YES) (NO) -- Yes very Would you recommend them to other veterans? (YES) (NO) -- Yes Comments: (Be Brief)